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Dimethoate Suppliers list
Company Name: Hangzhou FandaChem Co.,Ltd.
Tel: 008615858145714
Products Intro: Product Name:Dimethoate
Company Name: career henan chemical co
Tel: +86-371-86658258
Products Intro: Product Name: Dimethoate
CAS: 60-51-5
Purity:99% Package:1KG;7USD
Tel: +86 18953170293
Products Intro: Product Name:dimethoate
Company Name: Hubei Jusheng Technology Co.,Ltd.
Tel: 86-18871470254
Products Intro: Product Name:dimethoate
Purity:99% Package:5KG;1KG Remarks:C5H12NO3PS2
Company Name: Hubei xin bonus chemical co. LTD
Tel: 86-13657291602
Products Intro: Product Name:Dimethoate
Purity:0.99 Package:5KG;1KG

Dimethoate manufacturers

  • Dimethoate
  • $8.00 / KG
  • 2021-11-08
  • CAS:60-51-5
  • Min. Order: 100g
  • Purity: 99%
  • Supply Ability: 1000MT/Month
  • Dimethoate
  • $15.00-10.00 / KG
  • 2021-08-12
  • CAS:60-51-5
  • Min. Order: 1KG
  • Purity: 99%+ HPLC
  • Supply Ability: Monthly supply of 1 ton
  • Dimethoate
  • $7.00 / KG
  • 2019-07-06
  • CAS: 60-51-5
  • Min. Order: 1KG
  • Purity: 99%
  • Supply Ability: 1000KG

Related articles

  • What is Dimethoate?
  • Dimethoate is a systemic and contact insecticide–acaricide used on a range of insects including mites, flies, aphids, and plan....
  • Oct 11,2021
Dimethoate Chemical Properties
Melting point 52-52.5°C
Boiling point 107°C (0.05 torr)
density 1.281
vapor pressure 2.5 x 10-4 Pa (25 °C)
refractive index nD65 1.5334
Fp 107 °C
storage temp. 2-8°C
form solid
Water Solubility Slightly soluble. 2.5 g/100 mL
Merck 13,3246
BRN 1785339
CAS DataBase Reference60-51-5(CAS DataBase Reference)
NIST Chemistry ReferenceDimethoate(60-51-5)
EPA Substance Registry SystemDimethoate (60-51-5)
Safety Information
Hazard Codes Xn,T,F
Risk Statements 21/22-63-43-36/37/38-23/24/25-45-36-20/21/22-11-67-40
Safety Statements 2-36/37-24/25-23-53-26-16
WGK Germany 3
RTECS TE1750000
HazardClass 6.1(b)
PackingGroup III
HS Code 29309090
Hazardous Substances Data60-51-5(Hazardous Substances Data)
ToxicityLD50 orally in rats: 250 mg/kg (Schafer)
MSDS Information
Dimethoate Usage And Synthesis
DescriptionDimethoate is a grey-white crystalline solid at room temperature. It is sparingly soluble in water, soluble in methanol and cyclohexane, but very soluble in chloroform and benzene. It has been classified by the U.S. EPA under GUP. Dimethoate is used extensively for the control of crop pests such as mites, aphids, thrips, plant hoppers, white-flies, and a wide range of other insects that damage, crops, fruits, vegetables, and ornamental plants. Dimethoate is also used for the control of cattle grubs that infect livestock. Thermal decomposition of dimethoate is highly hazardous due to the release of fumes of dimethylsulphide, methyl mercaptan, carbon monoxide, carbon dioxide, phosphorus pentoxide, and nitrogen oxides.
References[1]Engenheiro, E. L., et al. "Influence of dimethoate on acetylcholinesterase activity and locomotor function in terrestrial isopods." Environmental Toxicology & Chemistry 24.3(2005):603.
[2]John, Susan, et al. "Protective effect of vitamin E in dimethoate and malathion induced oxidative stress in rat erythrocytes." The Journal of nutritional biochemistry 12.9 (2001): 500-504.
[3]Sharma, Yukti, et al. "Dimethoate-induced effects on antioxidant status of liver and brain of rats following subchronic exposure." Toxicology 215.3 (2005): 173-181.
DescriptionDimethoate is colorless crystalline substance, mp 49 ?C, bp 117 ?C/0.1 mm Hg, vp 0.25 mPa (25 ?C). The solubility in water is 23.8 g/L at 20 ?C and pH 7. It is readily soluble in polar organic solvents. Log Kow = 0.7. Dimethoate is relatively stable in aqueous media at pH 2–7 and hydrolyzed in alkaline media; DT50 (20 ?C) at pH 9 is 12 d.
Chemical PropertiesDimethoate is a gray-white crystalline solid at room temperature. It is sparingly soluble in water, soluble in methanol and cyclohexane, but very soluble in chloroform, benzene. It has been classifi ed by the US EPA under GUP. Dimethoate is used extensively for the control of crop pests, such as mites, aphids, thrips, plant-hoppers, white-fl ies, and a wide range of other insects that damage, crops, fruits, vegetables, and ornamental plants. Dimethoate is also used for the control of cattle grubs that infect livestock. Thermal decomposition of dimethoate is highly hazardous owing to the release of fumes of dimethylsulfi de, methyl mercaptane, carbon monoxide, carbon dioxide, phosphorus pentoxide, and nitrogenoxides.
UsesAn organophosphate insecticide. It is an anticholinesterase which disables cholinesterase, an enzyme essential for central nervous system function. Neurotoxic in humans.
Usesinsecticide, cholinesterase inhibitor
UsesSystemic and contact organophosphorus insecticide and acaricide used to control thrips and red spider mites on many agricultural crops, saw?ies on apples and plums, wheat bulb and olive ?ies.
UsesDimethoate is used to control of a wide range of insect and mite pests in many crops. It is also used to control flies in animal houses.
UsesSystemic and contact insecticide.
DefinitionChEBI: A monocarboxylic acid amide that is N-methylacetamide in which one of the hydrogens of the methyl group attached to the carbonyl moiety is replaced by a (dimethoxyphosphorothioyl)sulfanediyl group.
General DescriptionA white crystalline solid, with a camphor-like odor, white to grayish crystals for technical product. Dimethoate is a contact and systemic organophosphate insecticide effective against a broad range of insects and mites when applied on a wide range of crops. Dimethoate has not been produced in the U.S. since 1982.
Air & Water ReactionsDimethoate is stable in aqueous solution but is hydrolyzed by aqueous alkali.
Reactivity ProfileDimethoate is incompatible with alkaline preparations. Dimethoate is slightly corrosive to iron. Dimethoate is incompatible with sulfur based formulations. . Organophosphates are susceptible to formation of highly toxic and flammable phosphine gas in the presence of strong reducing agents such as hydrides. Partial oxidation by oxidizing agents may result in the release of toxic phosphorus oxides.
Health HazardVery toxic; the probable oral lethal dose in humans is between 50-500 mg/kg, or between 1 teaspoon and 1 ounce for a 70 kg (150 lb.) person. Dimethoate is a cholinesterase inhibitor, meaning it affects the central nervous system. Death is due to respiratory arrest arising from failure of respiratory center, paralysis of respiratory muscles, intense bronchoconstriction or all three.
Health HazardDimethoate is toxic to animals and humans. Occupational exposures cause poisoning with symptoms that include, but are not limited to, sweating, headache, weakness, giddiness, nausea, vomiting, stomach pains, blurred vision, pupillary constriction, slurred speech, and muscle twitching. Workers repeatedly exposed to dimethoate have shown symptoms of numbness, tingling sensations, incoordination, headache, dizziness, tremor, nausea, abdominal cramps, diffi culty breathing or respiratory depression, slow heart beat, and speech diffi culties. Prolonged exposures cause severe poisoning with adverse effects on the CNS, leading to incoordination, slurred speech, loss of refl exes, weakness, fatigue, involuntary muscle contractions, twitching, tremors of the tongue or eyelids, and eventually paralysis of the body extremities and the respiratory muscles, psychosis, irregular heart beats, unconsciousness, convulsions, coma, and death caused by respiratory failure or cardiac arrest.
Health HazardCholinesterase inhibitor; very toxic, exhibit-ing acute, delayed, and chronic toxicity;routes of entry—ingestion, skin contact,and inhalation; toxic symptoms include nausea, vomiting, diarrhea, excessive salivation,bronchoconstriction, and respiratory arrest;oral intake of 5–20 g may cause death toadult humans.
LD50 oral (mammal): 15 mg/kg
The oral LD50 in rat, however, is higher(within the range 150 mg/kg) than someother common organophosphorus insecti-cides, such as Nemaphos, Mevinphos, orCoumaphos.
Fire HazardAs with other organophosphorus pesticides, container may explode in heat of fire. The temperature of storage should not exceed 70-80F. Keep away from sources of heat, flames, or spark-generating equipment. Unstable in alkaline solution. Hydrolyzed by aqueous alkali. Stable in aqueous solutions. The compound is stable for 2 years under environmental conditions if stored in undamaged, original containers.
Agricultural UsesInsecticide, Miticide: Dimethoate is used to kill mites and insects systemically and on contact. It is used against a wide range of insects, including aphids, thrips, planthoppers, and whiteflies on ornamental plants, alfalfa, apples, corn, cotton, grapefruit, grapes, lemons, melons, oranges, pears, pecans, safflower, sorghum, soybeans, tangerines, tobacco, tomatoes, watermelons, wheat, and other vegetables. It is also used as a residual wall spray in farm buildings for house flies. Dimethoate has been administered to livestock for control of botflies. Dimethoate is available in aerosol spray, dust, emulsifiable concentrate, and ULV concentrate formulations.
Contact allergensThis organophosphorus compound is used as a contact and systemic insecticide and acaricide. It induced an erythema-multiform-like contact dermatitis in a ware- houseman in an agricultural consortium.
Safety ProfileA deadly human poison. Poison by ingestion, skin contact, intraperitoneal, and subcutaneous routes. Moderately toxic by intravenous route. Human systemic effects: coma, dyspnea,fasciculations. Questionable carcinogen with experimental carcinogenic data. Experimental teratogenic and reproductive effects. Human mutation data reported. When heated to decomposition it emits very toxic fumes of NOx、, POx, and SOx. See also ESTERS.
CarcinogenicityRats given diets with 0, 5, 25, or 100 ppm dimethoate (0, 0.25, 1.25, or 5 mg/kg/day) for 2 years showed dose-related trends for spleen hemangiosarcoma; combined spleen hemangioma and hemangiosarcoma; and combined spleen hemangioma, hemangiosarcoma, and skin hemangiosarcoma . There were also significant differences in pair-wise comparisons between controls and low-dose (0.25 mg/kg) or high-dose (5 mg/kg) groups for spleen (hemangioma/hemangiosarcoma) and combined tumors of spleen and skin hemangiosarcoma and lymph angioma/angiosarcoma; and between low and high doses for all tumors combined.
Environmental FateSoil. Duff and Menzer (1973) reported that in moist soils, dimethoate is converted to the oxygen analog, dimethoate carboxylic acid (dimethoxon) and two unidentified metabolites. The degradation rate of dimethoate in three different soils increased almost twofold with a 10°C increase in temperature (Kolbe et al., 1991). The reported half-lives of dimethoate in a humus-rich sandy soil, clay loam and heavy clay soil at 10 and 20°C are 15.3, 10.3, 15.5 days and 9.7, 4.8 and 8.5 days, respectively. Degradates included dimethoxon (O,O-dimethyl-S-(N-methylcarbamoylmethyl)phosphorothiolate) and unidentified polar compounds (Kolbe et al., 1991).
In a silt loam and sandy loam, reported Rf values were 0.42 and 0.45, respectively (Sharma et al., 1986).
Plant. In plants, oxidation/hydrolysis leads to the formation of the phosphorothioate. Other hydrolysis products in plants include O,O-dimethylphosphorodithioate and O,Odimethylphosphorophosphate which occurs via demethylation and hydrolytic clea
The half-life in Bermuda grass was reported to be 3.1 days (Beck et al., 1966). The disappearance half-lives of dimethoate on bean, tomato, cucumber and cotton plants were 4.3, 6.0, 3.8 and 3.3 days, respectively (Belal and Gomaa, 1979).
Surface Water. Though no products were identified, the half-life in raw river water was 8 weeks (Eichelberger and Lichtenberg, 1971). Photolytic. Dichlorvos should not undergo direct photolysis since it does not absorb UV light at wavele
Metabolic pathwayDimethoate is a systemic insecticide which is rapidly translocated to all parts of the plant. Metabolism to omethoate (dimethoate oxon), the active cholinesterase inhibitor, is a major pathway in both animals and plants. Degradative metabolism is either via dealkylation to yield both des-O- methyldimethoate and des-O-methylomethoate, followed by hydrolysis or dealkylation to give mono- and di-methyl phosphate, phosphorothioate and phosphorodithioate esters which are ultimately degraded to phosphate. An additional mechanism is through hydrolysis of the amide group to give dimethoate and omethoate carboxylic acids. An important additional metabolic pathway is oxidative N-demethylation of dimethoate and omethoate to give des-N-methyldimethoate and des-N-methy lomethoate via their respective N-hydroxymethyl derivatives. It is possible that the N-demethylation route could precede amidase action leading to dimethoate and omethoate carboxylic acids which are excreted in mammals. The 'thioglycolate' part of the molecule is metabolised to α-hydroxy-N-methylacetamide in mammals.
DegradationDimethoate is stable in aqueous media in the pH range 2-7 but hydrolysed under alkaline conditions (PM). Under acidic conditions the sole product of hydrolysis is desmethyldimethoate (2), whereas in alkali both desmethyldimethoate (2) and O,O-dimethylphosphorothioate (3) were formed. These products are in accord with other studies on the effects of pH on the hydrolysis of phospho-triesters, with dealkylation predominating under acidic conditions and SN2 attack on phosphorus by OH- at high pH (PSD, 1993).
Dimethoate is stable to photolysis. In an experiment to measure the degradation of an aqueous solution of dimethoate, a sample of [O-14C-methyl]dimethoate was irradiated with a xenon arc lamp for 15 days. The compound was extracted unchanged (93.4%) and no specific degradation products were identified (PSD, 1993).
Toxicity evaluationThe acute oral LD50 for rats is 387 mg/kg. Inhalation LC50 (4 h) for rats is >1.6 mg/L air. NOEL (2 y) for rats is 5 mg/kg diet (0.25 mg/kg/d). ADI is 2 μg/kg (sum of dimethoate and its oxon, i.e., omethoate). Oxidative desulfuration to form omethoate, the active AChE inhibitor, occurs both in mammals and plants. The main degradation routes are O-demethylation and amide hydrolysis that is important particularly for the selective species toxicity in animals. The cleavage of the P?S and S?C linkages also occurs to a considerable degree. Aerobic DT50 in soil is 2–4 d,whereas it was 22 d under anaerobic conditions.
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