| Identification | Back Directory | [Name]
LACTO-N-FUCOPENTAOSE III | [CAS]
25541-09-7 | [Synonyms]
LEX
LNF III
LNFP III
LNF III-GEL
CD15 ANTIGEN
LE(X)-LACTOSE
III3FUC-NLCOSE4
LEX-PENTASACCHARIDE
Lacto-N-fucopentaose Ⅲ
Lacto-N-fucopentaose Ⅲ
LEWIS-X PENTASACCHARIDE
LACTO-N-FUCOPENTAOSE III
LEWIS-X-PENTASACCHARIDE-GEL
LACTO-N-FUCOPENTAOSE III-GEL
Lacto-N-fucopentaose III ,95%
LACTO-N-FUCOPENTAOSE III, HUMAN MILK
lacto-N-fucopentaose iii*from human milk
GAL-BETA1,4(FUC-ALPHA1,3)GLCNAC-BETA1,3GAL-BETA1,4GLC
β-d-gal-(1→4)-[α-l-fuc-(1→3)]-β-d-glcnac-(1→3)-β-d-gal-(1→4)-d-glc
BETA-D-GAL-[1->4]-(ALPHA-L-FUC-[1->3])-BETA-D-GLCNAC-[1->3]-BETA-D-GAL-[1->4]-D-GLC
Lex-lactose, Lewis-X pentasaccharide, LNFP III, β-D-Gal-(1-4)-[α-L-Fuc-(1-3)]-β-D-GlcNAc-(1-3)-β-D-Gal-(1-4)-D-Glc
D-Glucose, O-6-deoxy-α-L-galactopyranosyl-(1→3)-O-[β-D-galactopyranosyl-(1→4)]-O-2-(acetylamino)-2-deoxy-β-D-glucopyranosyl-(1→3)-O-β-D-galactopyranosyl-(1→4)-
N-[2-[3,5-dihydroxy-2-(hydroxymethyl)-6-(1,2,4,5-tetrahydroxy-6-oxohexan-3-yl)oxyoxan-4-yl]oxy-6-(hydroxymethyl)-5-[3,4,5-trihydroxy-6-(hydroxymethyl)oxan-2-yl]oxy-4-(3,4,5-trihydroxy-6-methyloxan-2-yl)oxyoxan-3-yl]acetamide | [Molecular Formula]
C32H55NO25 | [MDL Number]
MFCD00214316 | [MOL File]
25541-09-7.mol | [Molecular Weight]
853.77 |
| Chemical Properties | Back Directory | [Boiling point ]
1264.6±65.0 °C(Predicted) | [density ]
1.71±0.1 g/cm3(Predicted) | [storage temp. ]
2-8°C | [pka]
12.39±0.20(Predicted) | [InChIKey]
CMQZRJBJDCVIEY-PVRYHJNXNA-N |
| Hazard Information | Back Directory | [Chemical Properties]
solid | [Uses]
Lacto-N-fucopentaose III (Cas# 25541-09-7) is a human milk oilgosaccharide. It can inhibit the adherence of pathogens to the intestinal epithelium. | [in vivo]
Lacto-N-fucopentaose III glycan (50 μg/mouse; i.v.; twice a week for 2 weeks) significantly reduces the severity of experimental autoimmune encephalomyelitis (EAE) and CNS inflammation, and skews peripheral immune response to a Th2 dominant profile in mice[1].
Lacto-N-fucopentaose III (35 μg dextran conjugate/mouse; s.c.; daily for 2 weeks) ameliorates acute and persisting hippocampal synaptic plasticity and transmission deficits in a Gulf War Illness mouse model[2]. |
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BOC Sciences
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