ChemicalBook--->CAS DataBase List--->42373-41-1

42373-41-1

42373-41-1 Structure

42373-41-1 Structure
IdentificationBack Directory
[Name]

ADENOSINE 5'-TRIPHOSPHATE POTASSIUM SALT
[CAS]

42373-41-1
[Synonyms]

ATP·K2
5'-ATP-K2
ATP dipotassium
ATP POTASSIUM SALT
ADENOSINE 5'-TRIPHOSPHATE POTASSIUM SALT
ADENOSINE-5'-TRIPHOSPHATE DIPOTASSIUM SALT
adenosine 5'-triphosphate dipotassium*from bacter
ADENOSINE 5'-TRIPHOSPHATE DIPOTASSIUMFRO M BACTERIA
Adenosine 5μ-triphosphate dihydrate dipotassium salt
Adenosine 5'-(tetrahydrogen triphosphate), dipotassiuM salt
Adenosine 5′-triphosphate dipotassium salt dihydrate,5′-ATP-K2
[Molecular Formula]

C10H14K2N5O13P3
[MDL Number]

MFCD00084672
[MOL File]

42373-41-1.mol
[Molecular Weight]

583.36
Chemical PropertiesBack Directory
[storage temp. ]

−20°C
[solubility ]

H2O: 50 mg/mL
[form ]

powder
[biological source]

microbial
[Water Solubility ]

H2O: 50mg/mL
Safety DataBack Directory
[Symbol(GHS) ]


GHS07
[Signal word ]

Warning
[Hazard statements ]

H315-H319-H335
[Precautionary statements ]

P261-P264-P271-P280-P302+P352-P305+P351+P338
[Hazard Codes ]

Xi
[Risk Statements ]

36/37/38
[Safety Statements ]

26-36
[WGK Germany ]

3
[F ]

10-21
Hazard InformationBack Directory
[Uses]

Adenosine 5′-triphosphate dipotassium salt hydrate has been used:
  • in in vitro transcription reaction as nucleotide source and in 2x RNA binding buffer
  • as a component of energy solution for calibrating crude cell extract
  • to test its dependency with PK11195 in mediating autophagy

[General Description]

Adenosine 5′-triphosphate (ATP) is synthesized in mitochondria by oxidative phosphorylation.
[Biochem/physiol Actions]

Adenosine 5′-triphosphate (ATP) is a central component of energy storage and metabolism in vivo. ATP is used in many cellular processes, respiration, biosynthetic reactions, motility, and cell division. ATP is a substrate of many kinases involved in cell signaling and of adenylate cyclase(s) that produce the second messenger?cAMP. ATP provides the metabolic energy to drive metabolic pumps. Metabolic disorders lead to ATP deficiency. Use of xanthine oxidoreductase inhibitors and inosine improves intracellular ATP levels.
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