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74341-78-9

74341-78-9 Structure

74341-78-9 Structure
IdentificationBack Directory
[Name]

3,4-Methylenedioxyethylamphetamine hydrochloride
[CAS]

74341-78-9
[Synonyms]

MDE-HCl
N-Ethyl-α-Methyl-
(±)-MDEA hydrochloride
3,4-MDEA (hydrochloride)
IBDIPBWIXJRJQM-UHFFFAOYSA-N
3,4-MDEA (hydrochloride) (CRM)
3,4-MDEA (hydrochloride) (exempt preparation)
MDEA hydrochloride, N-Ethyl-3,4-MDA hydrochloride
rac 3,4-Methylenedioxy-N-ethyl Amphetamine Hydrochloride
(±)-N-Ethyl-3,4-methylenedioxyamphetamine hydrochloride (MDEA)
3,4-Methylenedioxyethylamphetamine hydrochloride, MDE hydrochloride, MDE-HCl, MDEA hydrochloride
[Molecular Formula]

C12H18ClNO2
[MDL Number]

MFCD01659568
[MOL File]

74341-78-9.mol
[Molecular Weight]

243.73
Chemical PropertiesBack Directory
[Melting point ]

197-198° (Noggle); mp 201-202° (Braun)
[storage temp. ]

2-8°C
[solubility ]

DMF: 5 mg/ml; DMSO: 10 mg/ml; Ethanol: 0.3 mg/ml; PBS (pH 7.2): 5 mg/ml
[form ]

A neat solid
Safety DataBack Directory
[Hazard Codes ]

Xn,T,F
[Risk Statements ]

22-36/37/38-39/23/24/25-23/24/25-11
[Safety Statements ]

26-36/37/39-45-36/37-16-7
[WGK Germany ]

2
[RTECS ]

DF4914530
[Toxicity]

LD50 i.p. in mice: 102 mg/kg (Noggle)
Hazard InformationBack Directory
[Description]

3,4-MDEA (hydrochloride) (CRM) (Item No. ISO60196) is a certified reference material catagorized as an amphetamine. 3,4-MDEA (hydrochloride) (CRM) (Item No. ISO60196) is provided as a DEA exempt preparation. This product is intended for research and forensic applications.
[Uses]

3,4-Methylenedioxyethylamphetamine hydrochloride is a homologue of MDMA, a potential drugs of abuse. A component in Ecstasy street tablets. Controlled substance (hallucinogen).
[References]

[1] ROLAND W. FREUDENMANN  Manfred S. The Neuropsychopharmacology and Toxicology of 3,4-methylenedioxy-N-ethyl-amphetamine (MDEA)[J]. CNS drug reviews, 2006, 10 2: 89-116. DOI: 10.1111/j.1527-3458.2004.tb00007.x
[2] C M MILROY  A R F  J C Clark. Pathology of deaths associated with “ecstasy” and “eve” misuse.[J]. Journal of Clinical Pathology, 1996, 49 2: 149-153. DOI: 10.1136/jcp.49.2.149
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